The following excerpt can be found at this site:
http://dogtorj.tripod.com/id21.html
"In addition to successfully managing the epileptic, the dietary solution provides insight into the wide variety of presentations of this elusive condition. For instance, the typical age of onset in the dog ranges from six months to six years. If this is simply a genetic defect of some sort, why does that first seizure occur over such a wide range of ages? Also, why is it often a progressive condition in the afflicted individual, beginning with milder seizures and longer intervals but changing into more violent attacks that are closer together? This suggests that the cause of the seizures is getting worse over time, doesnt it? The idea that there is a lessening of the epileptics ability to handle the glutamate by a progressive deficiency in the reductase enzymes just makes sense. As the glue-induced malabsorption in the gut worsens, the individuals ability to process the rising glutamate in the brain becomes impaired by the dropping reductase levels. This explains both the variance in age of onset and the shrinking intervals between seizures. In addition, the attacks get more violent, especially once we institute anticonvulsant therapy thereby allowing the levels of glutamate to reach more toxic levels."
http://dogtorj.tripod.com/id21.html
"In addition to successfully managing the epileptic, the dietary solution provides insight into the wide variety of presentations of this elusive condition. For instance, the typical age of onset in the dog ranges from six months to six years. If this is simply a genetic defect of some sort, why does that first seizure occur over such a wide range of ages? Also, why is it often a progressive condition in the afflicted individual, beginning with milder seizures and longer intervals but changing into more violent attacks that are closer together? This suggests that the cause of the seizures is getting worse over time, doesnt it? The idea that there is a lessening of the epileptics ability to handle the glutamate by a progressive deficiency in the reductase enzymes just makes sense. As the glue-induced malabsorption in the gut worsens, the individuals ability to process the rising glutamate in the brain becomes impaired by the dropping reductase levels. This explains both the variance in age of onset and the shrinking intervals between seizures. In addition, the attacks get more violent, especially once we institute anticonvulsant therapy thereby allowing the levels of glutamate to reach more toxic levels."
2 comments:
Have you tried fasting or caloric restriction as an adjunct therapy? It is successful in nearly every other auotimmune disorder that I've researched thusfar. Could be the increased BDNF, increased DHEA, improved insulin sensitivity, or elimination of food allergens, but it's worth a shot if you haven't yet.
You should consider neurotransmitter testing in order to see where is the imbalance. There are inhibitory aminoacids you could try: taurine and 5-HTP being the safest, but there are others like glycine. I once saw a medical study that used glycine successfully to treat schizophrenia. You should talk to a GOOD doctor. I can't remember the link about glycine right now sorry.
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